acidosis

Shock , sepsis, acidosis , hypoxia, and so forth, are treated.

对休克,感染, 酸中毒, 缺氧等应加以治疗.

Chronic respiratory alkalosis ( CRA ) , CRA plus metabolic alkalosis and metabolic acidosis are seldom.

慢性呼吸性碱中毒 、 性呼吸性碱中毒并代谢性碱中毒和代谢性酸中毒是少见的.

A number of intoxicants are associated with metabolic acidosis.

许多毒性物质可引起代谢性酸中毒.

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

血液学研究显示有严重高磷血症 、 低钙血症和代谢性酸中毒.

Results CVVH, HDF and HD showed definite effects on severe lactic acidosis.

结果CVVH 、 hdF和hd治疗乳酸酸中毒疗效明确.

Methods: The animals achieved hypercapnic acidosis with mechanical hypoventilation before ischemia - reperfusion.

方法在缺血再灌注前,用低潮气量,低频率的机械通气方法造成动物的呼吸性酸中毒.

All patients presented with metabolic abnormalities, including hypoglycemia, and lactic acidosis.

所有这些病人都表现出代谢异常, 包括低血糖血症和乳酸性酸中毒.

Metabolic acidosis was resolved by both hemoglobin solutions, but not by dextran.

两组均无代谢性酸中毒, 而右旋糖酐组有.

Retinal NV induced by acidosis relates to VEGF.

酸中毒引起的视网膜新生血管与VEGF有关.

Methanol can cause poisoning, systemic acidosis, optic nerve damage and central nervous system ( CNS ) effects.

甲醇能造成中毒, 组织酸毒症, 选择性神经伤害和影响中枢神经.

Hypercalciuria was considered as a secondary condition when ( It was ) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒,就可以认为是继发病.

Respiratory acidosis is caused by retention of CO 2 secondary to decreased alveolar ventilation.

呼吸性酸中毒是由继发于肺泡通气功能降低引起的CO2贮留所致.

Biochemical investigations showed hyponatremia , hyperkalemia, hypochloremia, metabolic acidosis and large amount of urinary sodium excretion.

经过适当治疗和水分补充,临床肠胃症状很快获得缓解.

Risk factors for AGML low blood pressure , damage of liver function, serious infection, acidosis and uremia.

临床分析提示,低血压 、 功能损害 、 重感染 、 中毒、毒症等是急性胃粘膜病变的危险因素.

RESULTS: Hemorrhagic shock reduced functional capillary density ( FCD ) by 70 % and caused significant metabolic acidosis.

结果: 失血性休克使毛细血管密度 ( FCD ) 减少70%,引起显著的代谢性酸中毒.